Myeloperoxidase (MPO) delays neutrophil apoptosis and prolongs acute pulmonary inflammation

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Myeloperoxidase delays neutrophil apoptosis through CD11b/CD18 integrins and prolongs inflammation.

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Myeloperoxidase (MPO) gene mutation in hereditary MPO deficiency.

Myeloperoxidase (MPO), present in the azurophilic granules of polymorphonuclear leukocytes, is a myeloid enzyme whose synthesis is restricted to promyelocytes. Complete hereditary MPO deficiency affects 1 in 2,000 to 4,000 individuals; however, the genetic cause of this defect is unclear. We have determined the molecular basis of MPO deficiency in one individual (SQ). Granulocytes of SQ had no ...

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ژورنال

عنوان ژورنال: The FASEB Journal

سال: 2008

ISSN: 0892-6638,1530-6860

DOI: 10.1096/fasebj.22.1_supplement.328.6